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Nature Reviews: Drug Discovery Article Describes Design Of Glycomimetic Compounds And Newly "Druggable" Disease Targets
GlycoMimetics, Inc. announced the publication of an article in Nature Reviews: Drug Discovery describing how novel small molecule compounds that mimic carbohydrates can be successfully designed and optimized to treat cancer, inflammatory disorders and other diseases.
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Good Fences Make Good Neighbors
Our genome is a patchwork of neighborhoods that couldn"t be more different: Some areas are hustling and bustling with gene activity, while others are sparsely populated and in perpetual lock-down. Breaking down just a few of the molecular fences that separate them blurs the lines and leads to the inactivation of at least two tumor suppressor genes, according to researchers at the Salk Institute for Biological Studies.
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Key To Improving Prognosis In Acute Heart Failure - Better Treatment Selection And Improved Therapies
Today, acute heart failure represents the most common reason for hospitalisation in the over-65 population. Although hospital care improves symptoms in the first 24 hours after admission in around 50% of these patients, acute heart failure events still remain associated with a more than 50% mortality and rehospitalisation rate at 6-12 months. "Indeed," says Professor Marco Metra from the Cardiology Department of the University of Brescia, Italy, "it is the very rapid onset of symptoms and the need for urgent therapy which characterise the condition."1,2
Public Health

The Immune Response To Influenza Virus Isn't 'All Good'

Complications following infection with the virus that causes flu (influenza virus) are one of the top ten causes of death in the United States. Although infection with influenza virus can directly cause death, many deaths following infection with influenza virus occur because the individual develops pneumonia due to secondary infection with bacteria such as Streptococcus pneumoniae. How influenza makes individuals more sensitive to pneumonia-causing secondary bacterial infections is not well understood. However, Jane Deng and colleagues, at the University of California, Los Angeles, have now determined, through studies in mice, one mechanism by which influenza might sensitize individuals to secondary bacterial pneumonia. In the study, it was found that molecules known as type I IFNs, which are key mediators of the antiviral immune response initiated by infection with influenza virus, impaired the ability of mice to mount an adequate immune response to subsequent pneumonia-causing bacterial infection. In particular, the type I IFNs decreased production of soluble factors that attract neutrophils, immune cells central to the initial antibacterial immune response, to sites of bacterial infection. The authors therefore suggest that the pathway uncovered in their study might provide a new avenue of research for those developing ways to combat pneumonia following infection with influenza virus. TITLE: Type I IFNs mediate development of postinfluenza bacterial pneumonia in mice https://www.the-jci.org/article.php?id=35412 AUTHOR CONTACT: Jane C. Deng David Geffen School of Medicine, UCLA, Los Angeles, California, USA. Karen Honey Journal of Clinical Investigation


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